Difference between revisions of "Category:Hepatotoxicity, Acute"
Jump to navigation
Jump to search
Line 3: | Line 3: | ||
|pagebody = <div style="text-align: left; direction: ltr; margin-left: 1em;"> | |pagebody = <div style="text-align: left; direction: ltr; margin-left: 1em;"> | ||
'''Acute hepatotoxicity''' almost invariably causes widespread haemorrhages in the body due to excessive consumption of the clotting factors in the damaged [[Liver - Anatomy & Physiology|liver]] coupled with failure to produce these factors by the damaged [[Liver - Anatomy & Physiology|liver]]. | '''Acute hepatotoxicity''' almost invariably causes widespread haemorrhages in the body due to excessive consumption of the clotting factors in the damaged [[Liver - Anatomy & Physiology|liver]] coupled with failure to produce these factors by the damaged [[Liver - Anatomy & Physiology|liver]]. | ||
− | + | NB: former use of carbon tetrachloride as an anthelmintic and phosphorous as a rodenticide has produced [[Liver - Anatomy & Physiology|liver]] necrosis. | |
</div> | </div> | ||
|contenttitle =Content | |contenttitle =Content |
Revision as of 22:42, 7 June 2010
Hepatotoxicity, Acute
Acute hepatotoxicity almost invariably causes widespread haemorrhages in the body due to excessive consumption of the clotting factors in the damaged liver coupled with failure to produce these factors by the damaged liver. NB: former use of carbon tetrachloride as an anthelmintic and phosphorous as a rodenticide has produced liver necrosis.
Pages in category "Hepatotoxicity, Acute"
The following 3 pages are in this category, out of 3 total.