Changes

The life cycle of ''T. axei'' is typical of a trichostrongyloid, and is direct.  The eggs develop into infective L3 in around one week.  The larvae are then ingested by the host, and the larvae become exsheathed.  They penetrate the mucosa of the small intestine.  There are two further moults, and then the larve become adults.  

The life cycle of ''T. axei'' is typical of a trichostrongyloid, and is direct.  The eggs develop into infective L3 in around one week.  The larvae are then ingested by the host, and the larvae become exsheathed.  They penetrate the mucosa of the small intestine.  There are two further moults, and then the larve become adults.  

==== Pathogenicity ====

==== Pathogenicity ====

*Similar to ''Ostertagia'', except L3 penetrate mucosa between gastric glands. Leads to hyperplastic nodular lesions (several cms diameter) and erosions where epithelium is lost. Not usually a primary pathogen in horses.

Similar to ''Ostertagia'', except L3 penetrate mucosa between gastric glands. Leads to hyperplastic nodular lesions (several cms diameter) and erosions where epithelium is lost. Not usually a primary pathogen in horses.

 

 

[[Category:Trichostrongyloidea]][[Category:Horse_Nematodes]]

[[Category:Trichostrongyloidea]][[Category:Horse_Nematodes]]

[[Category:To_Do_-_Max]]

[[Category:To_Do_-_Max]]