Difference between revisions of "Adrenogenital Syndrome"
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|linktext =Reproductive System | |linktext =Reproductive System | ||
|maplink = Reproductive System (Content Map) - Anatomy & Physiology | |maplink = Reproductive System (Content Map) - Anatomy & Physiology | ||
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|sublink1=Reproductive System - Anatomy & Physiology#Fertilisation.2C Implantation and Early Embryonic Development | |sublink1=Reproductive System - Anatomy & Physiology#Fertilisation.2C Implantation and Early Embryonic Development | ||
|subtext1=FERTILISATION , IMPLANTATION AND EARLY EMBRYONIC DEVELOPMENT | |subtext1=FERTILISATION , IMPLANTATION AND EARLY EMBRYONIC DEVELOPMENT | ||
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* Appears male with a '''micropenis and scrotum''' | * Appears male with a '''micropenis and scrotum''' | ||
* Both male and female internal genitalia | * Both male and female internal genitalia | ||
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| + | [[Category:Reproductive System]] | ||
Revision as of 15:06, 22 August 2010
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Aetiology
- Genotype is XX as should be female
- Ovaries develop normally
- Foetal Adrenal Gland is hyperactive and secretes large quantities of Adrenal Androgens, due to a 21 hydroxylase mutation.
Consequence
- Wolffian Ducts persist due to the presence of Androgens, thus allowing development of male internal and external genitalia.
- Mullarian Ducts persist due to the absence of testes, hence absence of Sertoli cell MIH secretion. This allows formation of female internal genitalia.
Morphology
- Appears male with a micropenis and scrotum
- Both male and female internal genitalia