Difference between revisions of "Adrenogenital Syndrome"
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| + | |linkpage =Reproductive System - Anatomy & Physiology | ||
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| + | |maplink = Reproductive System (Content Map) - Anatomy & Physiology | ||
| + | |sublink1=Reproductive System - Anatomy & Physiology#Fertilisation.2C Implantation and Early Embryonic Development | ||
| + | |subtext1=FERTILISATION , IMPLANTATION AND EARLY EMBRYONIC DEVELOPMENT | ||
| + | |sublink2=Fertilisation, Implantation and Early Embryonic Development -Sexual Differentiation- Anatomy & Physiology | ||
| + | |subtext2=SEXUAL DIFFERENTIATION | ||
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== Aetiology == | == Aetiology == | ||
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Revision as of 15:21, 29 November 2010
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Aetiology
- Genotype is XX as should be female
- Ovaries develop normally
- Foetal Adrenal Gland is hyperactive and secretes large quantities of Adrenal Androgens, due to a 21 hydroxylase mutation.
Consequence
- Wolffian Ducts persist due to the presence of Androgens, thus allowing development of male internal and external genitalia.
- Mullarian Ducts persist due to the absence of testes, hence absence of Sertoli cell MIH secretion. This allows formation of female internal genitalia.
Morphology
- Appears male with a micropenis and scrotum
- Both male and female internal genitalia