Difference between revisions of "Physeal Dysplasia with Slipped Capital Femoral Epiphysis"
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*Most common type of physeal fracture in small animals and the proximal femur is the most common site | *Most common type of physeal fracture in small animals and the proximal femur is the most common site | ||
*In horses, physeal dysplasia is synonymus with [[Physitis|physitis]] | *In horses, physeal dysplasia is synonymus with [[Physitis|physitis]] | ||
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| + | '''More about Physeal dysplasia''' | ||
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| + | *Also seen in the coypu with minimal trauma | ||
| + | *Femoral head physes contribute 30-40% of longitudinal growth of the femur in small animals | ||
| + | *Slipped epiphysis -> Salter-Harris type I where only the physis is involved | ||
| + | **Most common type of physeal fracture in small animals and the proximal femur is the most common site | ||
| + | **In traumatic cases, the fracture crosses through mutiple zones of the physis, with chondrocytes maintaining their linear arrangement on both sides of the fracture site for 1-4d following trauma | ||
| + | *Slipped capital femoral epiphysis – seen in humans, mainly adolescent boys, autosominal dominant with variable penetrance | ||
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| + | In cats: | ||
| + | *'''[Physeal dysplasia with slipped capital femoral epiphysis in 13 cats. Craig LE. Vet Path (2001) 38 92-7]''' | ||
| + | **Age 12-21months | ||
| + | **90% males | ||
| + | **Often obese, lots of Siamese cats | ||
| + | **Normal growth plates close at 7-9mths so these physes should have been closed | ||
| + | **Histology – physeal dysplasia with persistence of an open, disorganized growth plate | ||
| + | ***Wide physis, necrotic cartilage at the cleavage site | ||
| + | ***Irregular clusters of chondrocytes separated by abundant chondroid matrix. Similar lesions in femoral neck. | ||
| + | **No necrosis of the femoral head, although there is usually a small areas of peripheral attachment at the perichondral fibrocartilagenous complex – could provide some vacularization **The ligamentum teres provides no blood supply to the femoral head | ||
| + | **Clusters of chondrocytes present on either side of the cleavage site | ||
| + | **Differs from [[Joints Developmental - Pathology#Osteochondrosis|osteochondrosis]] by: | ||
| + | ***More diffuse affecting the whole physis (in OC – focal failure of endochondral ossification) | ||
| + | ***Chondrocytes are arranged in disorganized clusters rather than parallel rows | ||
| + | ***The cat breeds have not been selected for rapid growth | ||
| + | **Insulin may have a role to play – most of the cats were obese....Similar deal to horse osteochondrosis | ||
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[[Category:Bones - Degenerative Pathology]] | [[Category:Bones - Degenerative Pathology]] | ||
Latest revision as of 12:02, 3 March 2011
- Slipped capital femoral epiphysis seen in dogs, foals and calves
- Associated with severe trauma
- Pigs – manifestation of osteochondrosis with only minimal trauma required
- Most common type of physeal fracture in small animals and the proximal femur is the most common site
- In horses, physeal dysplasia is synonymus with physitis
More about Physeal dysplasia
- Also seen in the coypu with minimal trauma
- Femoral head physes contribute 30-40% of longitudinal growth of the femur in small animals
- Slipped epiphysis -> Salter-Harris type I where only the physis is involved
- Most common type of physeal fracture in small animals and the proximal femur is the most common site
- In traumatic cases, the fracture crosses through mutiple zones of the physis, with chondrocytes maintaining their linear arrangement on both sides of the fracture site for 1-4d following trauma
- Slipped capital femoral epiphysis – seen in humans, mainly adolescent boys, autosominal dominant with variable penetrance
In cats:
- [Physeal dysplasia with slipped capital femoral epiphysis in 13 cats. Craig LE. Vet Path (2001) 38 92-7]
- Age 12-21months
- 90% males
- Often obese, lots of Siamese cats
- Normal growth plates close at 7-9mths so these physes should have been closed
- Histology – physeal dysplasia with persistence of an open, disorganized growth plate
- Wide physis, necrotic cartilage at the cleavage site
- Irregular clusters of chondrocytes separated by abundant chondroid matrix. Similar lesions in femoral neck.
- No necrosis of the femoral head, although there is usually a small areas of peripheral attachment at the perichondral fibrocartilagenous complex – could provide some vacularization **The ligamentum teres provides no blood supply to the femoral head
- Clusters of chondrocytes present on either side of the cleavage site
- Differs from osteochondrosis by:
- More diffuse affecting the whole physis (in OC – focal failure of endochondral ossification)
- Chondrocytes are arranged in disorganized clusters rather than parallel rows
- The cat breeds have not been selected for rapid growth
- Insulin may have a role to play – most of the cats were obese....Similar deal to horse osteochondrosis