Difference between revisions of "Immunity to Fungi"
Jump to navigation
Jump to search
m (Text replace - 'Macrophages - WikiBlood' to 'Macrophages') |
|||
| Line 2: | Line 2: | ||
*[[T cell differentiation - WikiBlood|T cell]] immunity- it is thought that [[T cell differentiation - WikiBlood#TH1 Cells|helper T cells]] release cytokines to activate [[Macrophages|macrophages]] to fight the infection | *[[T cell differentiation - WikiBlood|T cell]] immunity- it is thought that [[T cell differentiation - WikiBlood#TH1 Cells|helper T cells]] release cytokines to activate [[Macrophages|macrophages]] to fight the infection | ||
*[[Neutrophils|Neutrophils]]- there is now growing evidence for the role of [[Neutrophils|neutrophils]] in some infections of the respiratory system, with defensin and the nitric oxide pathways implicated[[Category:Fungi]] | *[[Neutrophils|Neutrophils]]- there is now growing evidence for the role of [[Neutrophils|neutrophils]] in some infections of the respiratory system, with defensin and the nitric oxide pathways implicated[[Category:Fungi]] | ||
| + | [[Category:To_Do_-_Fungi]] | ||
Revision as of 11:32, 25 June 2010
Cutaneous and superficial fungal infections are normally self-limiting, with recovery associated with a resistance to re-infection. As hosts often develop delayed-type hypersensitivity, this resistance appears to be cell-mediated.
- T cell immunity- it is thought that helper T cells release cytokines to activate macrophages to fight the infection
- Neutrophils- there is now growing evidence for the role of neutrophils in some infections of the respiratory system, with defensin and the nitric oxide pathways implicated