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Text replace - 'Macrophages - WikiBlood' to 'Macrophages'
Cutaneous and superficial fungal infections are normally self-limiting, with recovery associated with a resistance to re-infection. As hosts often develop delayed-type [[Hypersensitivity - WikiBlood|hypersensitivity]], this resistance appears to be cell-mediated.
Cutaneous and superficial fungal infections are normally self-limiting, with recovery associated with a resistance to re-infection. As hosts often develop delayed-type [[Hypersensitivity - WikiBlood|hypersensitivity]], this resistance appears to be cell-mediated.
*[[T cell differentiation - WikiBlood|T cell]] immunity- it is thought that [[T cell differentiation - WikiBlood#TH1 Cells|helper T cells]] release cytokines to activate [[Macrophages - WikiBlood|macrophages]] to fight the infection
*[[T cell differentiation - WikiBlood|T cell]] immunity- it is thought that [[T cell differentiation - WikiBlood#TH1 Cells|helper T cells]] release cytokines to activate [[Macrophages|macrophages]] to fight the infection
*[[Neutrophils|Neutrophils]]- there is now growing evidence for the role of [[Neutrophils|neutrophils]] in some infections of the respiratory system, with defensin and the nitric oxide pathways implicated[[Category:Fungi]]
*[[Neutrophils|Neutrophils]]- there is now growing evidence for the role of [[Neutrophils|neutrophils]] in some infections of the respiratory system, with defensin and the nitric oxide pathways implicated[[Category:Fungi]]