Changes

Cutaneous and superficial fungal infections are normally self-limiting, with recovery associated with a resistance to re-infection. As hosts often develop delayed-type [[Hypersensitivity - WikiBlood|hypersensitivity]], this resistance appears to be cell-mediated.

Cutaneous and superficial fungal infections are normally self-limiting, with recovery associated with a resistance to re-infection. As hosts often develop delayed-type [[Hypersensitivity - WikiBlood|hypersensitivity]], this resistance appears to be cell-mediated.

*[[T cell differentiation - WikiBlood|T cell]] immunity- it is thought that [[T cell differentiation - WikiBlood#TH1 Cells|helper T cells]] release cytokines to activate [[Macrophages - WikiBlood|macrophages]] to fight the infection

*[[T cell differentiation - WikiBlood|T cell]] immunity- it is thought that [[T cell differentiation - WikiBlood#TH1 Cells|helper T cells]] release cytokines to activate [[Macrophages - WikiBlood|macrophages]] to fight the infection

*[[Neutrophils - WikiBlood|Neutrophils]]- there is now growing evidence for the role of [[Neutrophils - WikiBlood|neutrophils]] in some infections of the respiratory system, with defensin and the nitric oxide pathways implicated[[Category:Fungi]]

*[[Neutrophils|Neutrophils]]- there is now growing evidence for the role of [[Neutrophils|neutrophils]] in some infections of the respiratory system, with defensin and the nitric oxide pathways implicated[[Category:Fungi]]