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==Gastric Causes of Colic==
 
==Gastric Causes of Colic==
*[[Gastric Ulceration - Horse|'''Gastroduodenal ulceration''' or ''''Equine Gastric Ulcer Syndrome (EGUS)'''']] is a common cause of mild to moderate colic.
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*[[Gastric Ulceration - Horse|'''Gastroduodenal ulceration''' or ''''Equine Gastric Ulcer Syndrome (EGUS)'''']]
 
*'''Pyloric obstruction and delayed gastric emptying:''' pyloric stenosis is a structural resistance to gastric outflow.  Congenital pyloric stenosis has been reported in foals and one yearling and results from hypertrophy of the pylroic musculature (135-137).  Aquired pyloric stenosis can result form neoplasia or duodenal ulceration(138-141).  Clinical signs depend on the degree of obstruction but include abodminal pain, salivation and bruxism.  Complete or near-complete obstruction can result in gastric reflux and reflux oesophagitis.  In foals with congenital pyloric hypertrophy, clinical signs may begin with the consumption of solid feed.  In foals a presumtpive diagnosis can be made with gastric endoscopy and radiography (plain and contrast studies).  Depending on the cause and severity of disease, gastric endoscopy may provide a presumptive diagnosis in the adult horse.  Measurement of gastric emptying can aid the diagnosis.  Several methods of measurement are currently available, including nuclear scintigraphy, acetaminophen absorption, and postconsumptionn[13C]-octanoic acid blood or breath testing (130,142,143).  During an exploratory laparotomy, a distended stomach and thickened pylorus  are accompanied by a reltively empty intestinal tract.  If complete obstruction is not present, medical theray with a prokinetic such as bethanechol can increase the rate of gastric emptying(130).  Phenylbutazone and cisapride have also been shown to attenuate the delay in gastric emptying caused by endotoxin administration(142,244).  Surgical repair is necessary for definitive treatment of complete or near-complete obstruction and consists of either gastroenterostomy or pyloroplasty(84,131).  Pyloric duodenal intussusception has been reported in an adult horse with colic(145).
 
*'''Pyloric obstruction and delayed gastric emptying:''' pyloric stenosis is a structural resistance to gastric outflow.  Congenital pyloric stenosis has been reported in foals and one yearling and results from hypertrophy of the pylroic musculature (135-137).  Aquired pyloric stenosis can result form neoplasia or duodenal ulceration(138-141).  Clinical signs depend on the degree of obstruction but include abodminal pain, salivation and bruxism.  Complete or near-complete obstruction can result in gastric reflux and reflux oesophagitis.  In foals with congenital pyloric hypertrophy, clinical signs may begin with the consumption of solid feed.  In foals a presumtpive diagnosis can be made with gastric endoscopy and radiography (plain and contrast studies).  Depending on the cause and severity of disease, gastric endoscopy may provide a presumptive diagnosis in the adult horse.  Measurement of gastric emptying can aid the diagnosis.  Several methods of measurement are currently available, including nuclear scintigraphy, acetaminophen absorption, and postconsumptionn[13C]-octanoic acid blood or breath testing (130,142,143).  During an exploratory laparotomy, a distended stomach and thickened pylorus  are accompanied by a reltively empty intestinal tract.  If complete obstruction is not present, medical theray with a prokinetic such as bethanechol can increase the rate of gastric emptying(130).  Phenylbutazone and cisapride have also been shown to attenuate the delay in gastric emptying caused by endotoxin administration(142,244).  Surgical repair is necessary for definitive treatment of complete or near-complete obstruction and consists of either gastroenterostomy or pyloroplasty(84,131).  Pyloric duodenal intussusception has been reported in an adult horse with colic(145).
 
*'''[[Gastric Dilation and Rupture - Horse|Gastric dilation and rupture]]'''
 
*'''[[Gastric Dilation and Rupture - Horse|Gastric dilation and rupture]]'''
*'''[[Gastric Impaction - Horse|Gastric impaction]]''' can cause mild, insidious abdominal pain or acute, severe colic.
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*'''[[Gastric Impaction - Horse|Gastric impaction]]'''
 
*'''Food Engorgement:''' a life threatening condition, with a great risk of gastric rupture, and also of secondary [[laminitis]].  It is caused by excess food intake, for example, a horse that has broken into a food store.
 
*'''Food Engorgement:''' a life threatening condition, with a great risk of gastric rupture, and also of secondary [[laminitis]].  It is caused by excess food intake, for example, a horse that has broken into a food store.
 
*'''Gastric/pyloric spasm:''' this commonly affects racehorses, immediately after racing, and is known colloquially as 'racehorse colic'.  Typically, the animal will have had access to cold water, but this is not always the case.  Although the signs of colic seen may be very violent, this condition is not associated with any risk of gastric rupture.  '''Spasmolytic''' drugs are ineffective in treatment, however, naso-gastric intubation is immediately curative.
 
*'''Gastric/pyloric spasm:''' this commonly affects racehorses, immediately after racing, and is known colloquially as 'racehorse colic'.  Typically, the animal will have had access to cold water, but this is not always the case.  Although the signs of colic seen may be very violent, this condition is not associated with any risk of gastric rupture.  '''Spasmolytic''' drugs are ineffective in treatment, however, naso-gastric intubation is immediately curative.
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