Difference between revisions of "Vitamin K Deficiency"
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| + | ==Introduction== | ||
The absolute or relative deficiency of vitamin K can give rise to defective coagulation. | The absolute or relative deficiency of vitamin K can give rise to defective coagulation. | ||
Anticoagulant rodenticide toxiticy is one of the most common causes of acquired coagulopathy in small animals. Warfarin itself has a short half-life and a fairly low toxicity in non-rodent species, so unless large or repeated doses are consumed clinical bleeding is rare. However, the second generation anticoagulant rodenticides are far more potent, and it is possible for a domestic animal to acquire secondary poisoning by ingesting a killed rodent<sup>1</sup>. | Anticoagulant rodenticide toxiticy is one of the most common causes of acquired coagulopathy in small animals. Warfarin itself has a short half-life and a fairly low toxicity in non-rodent species, so unless large or repeated doses are consumed clinical bleeding is rare. However, the second generation anticoagulant rodenticides are far more potent, and it is possible for a domestic animal to acquire secondary poisoning by ingesting a killed rodent<sup>1</sup>. | ||
| − | + | ==References== | |
Also see [[Anticoagulant Rodenticide Toxicity]] | Also see [[Anticoagulant Rodenticide Toxicity]] | ||
[[Category:Coagulation Defects]] | [[Category:Coagulation Defects]] | ||
[[Category:To Do - Blood]][[Category:To Do - Clinical]] | [[Category:To Do - Blood]][[Category:To Do - Clinical]] | ||
Revision as of 18:30, 2 October 2010
Introduction
The absolute or relative deficiency of vitamin K can give rise to defective coagulation. Anticoagulant rodenticide toxiticy is one of the most common causes of acquired coagulopathy in small animals. Warfarin itself has a short half-life and a fairly low toxicity in non-rodent species, so unless large or repeated doses are consumed clinical bleeding is rare. However, the second generation anticoagulant rodenticides are far more potent, and it is possible for a domestic animal to acquire secondary poisoning by ingesting a killed rodent1.
References
Also see Anticoagulant Rodenticide Toxicity