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Clostridium species (view source)

Revision as of 10:10, 26 May 2008

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**Fluid in the peritoneal cavity and pericardial sac due to increased capillary permeability

*''C. perfringens'' type C:

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**Acute enterotoxaemia in adult sheep, 'struck'

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**Sudden death or terminal convulsions in sheep at pasture

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**Beta toxin plays major role in pathogenesis of the disease

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**Post mortem: jejunal ulceration; hyperaemia in small intestine; fluid accumulation in peritoneal cavity; congestion of peritoneal vessels; petechial haemorrhages

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**

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*''C. perfringens'' type D:

**[[Intestines - Catarrhal Enteritis#"Pulpy Kidney" Disease|Pulpy kidney disease]] in well-fed 3-10 week-old lambs

**Follows overeating high grain diet or luchious pasture

**Starch from partially digested food enterering the intestine from the rumen allows rapid clostridial proliferation

**Epsilon toxin activated by proteolytic enzymes causes toxaemia

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**Lambs found dead or with opisthotonos, convulsions, coma

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**Lambs found dead or with opisthotonos, convulsions, coma in acute phases

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**Blindness and head pressing in subacute disease; bloat in later stages

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**Hyperglycaemia, glycosuria

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**Post mortem: hyperaemia in intestine; fluid in pericardial sac; kidney autolysis with pulpy cortical softening (acute death)

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**Subacute death causes symmetrical encephalomalacia and haemorrhage in basal ganglia and midbrain

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** [[Intestines - Fibrinous/ Haemorrhagic Enteritis#Colitis X|Colitis X]]

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*** ''C. perfringens'' type D only.

** [[Peritoneal cavity - inflammatory#In cattle|Peritonitis in cattle]]

** [[Respiratory system - clinical signs#Dysphagia|Dysphagia in horses]]

Flawrence

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