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Created page with '===Clinical=== * Catarrhal gastritis can be fatal since it makes the animal vomit and can produce rapid dehydratio…'
===Clinical===
* Catarrhal gastritis can be fatal since it makes the animal [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex|vomit]] and can produce rapid dehydration.
** May die in day or two if [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex|vomiting]] is persistent and untreated.
* Extracellular fluid (isontonic) is lost, and so blood very quickly becomes viscous.
** Death may occur from hypovolaemic shock
*** Particularly in young animals (can be very quick).
===Pathology===
* The mucosa appears swollen and hyperaemic, with thickened rugae.
** Mild inflammation, hyperaemia, and oedema
** Infiltration of inflammatory cells
** No fibrin or haemorrhage.
* The surface of the mucosa is covered by a white, sticky catarrhal exudate which lines the [[Forestomach - Anatomy & Physiology|stomach]].
===Pathogenesis===
* There are numerous causes of catarrhal gastritis
*# Ingestion of mild irritant
*# Systemic bacterial diseases
*# Infectious enteric diseases e.g.
*#* Transmissible gastro enteritis (TGE)
*#* E.coli
*#* Salmonella etc.
* Dogs are very prone catarrhal gastritis.
** Anything that affects dogs tends to produce [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex|vomiting]].
** Dogs eat almost anything and this can make them [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex|vomit]] for a short time.
[[Category:Stomach and Abomasum - Inflammatory Pathology]][[Category:Dog]]
* Catarrhal gastritis can be fatal since it makes the animal [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex|vomit]] and can produce rapid dehydration.
** May die in day or two if [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex|vomiting]] is persistent and untreated.
* Extracellular fluid (isontonic) is lost, and so blood very quickly becomes viscous.
** Death may occur from hypovolaemic shock
*** Particularly in young animals (can be very quick).
===Pathology===
* The mucosa appears swollen and hyperaemic, with thickened rugae.
** Mild inflammation, hyperaemia, and oedema
** Infiltration of inflammatory cells
** No fibrin or haemorrhage.
* The surface of the mucosa is covered by a white, sticky catarrhal exudate which lines the [[Forestomach - Anatomy & Physiology|stomach]].
===Pathogenesis===
* There are numerous causes of catarrhal gastritis
*# Ingestion of mild irritant
*# Systemic bacterial diseases
*# Infectious enteric diseases e.g.
*#* Transmissible gastro enteritis (TGE)
*#* E.coli
*#* Salmonella etc.
* Dogs are very prone catarrhal gastritis.
** Anything that affects dogs tends to produce [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex|vomiting]].
** Dogs eat almost anything and this can make them [[Control of Feeding - Anatomy & Physiology#The Vomit Reflex|vomit]] for a short time.
[[Category:Stomach and Abomasum - Inflammatory Pathology]][[Category:Dog]]