Changes

===Pathogenesis===

===Pathogenesis===

There are two manifestations of [[The Abomasum - Anatomy & Physiology|abomasal]] displacemet. In both the [[The Abomasum - Anatomy & Physiology|abomasum]] becomes trapped between [[The Rumen - Anatomy & Physiology|rumen]] and abdominal wall. The more comon presentation is the left displacement (LDA) which is Ventral and to the left of the rumen. The [[The Omasum - Anatomy & Physiology|omasum]], [[The Reticulum - Anatomy & Physiology|reticulum]] and [[Liver - Anatomy & Physiology|liver]] are also displaced.

There are two manifestations of [[The Abomasum - Anatomy & Physiology|abomasal]] displacemet. In both the [[The Abomasum - Anatomy & Physiology|abomasum]] becomes trapped between [[Rumen - Anatomy & Physiology|rumen]] and abdominal wall. The more comon presentation is the left displacement (LDA) which is Ventral and to the left of the rumen. The [[The Omasum - Anatomy & Physiology|omasum]], [[The Reticulum - Anatomy & Physiology|reticulum]] and [[Liver - Anatomy & Physiology|liver]] are also displaced.

Abomasal atony and increased gas production leads to displacement. Factors reducing abomasal motility include a high concentrate diet, increased [[Volatile Fatty Acids - Anatomy & Physioogy|volatile fatty acids]] from the rumen and periparturient disease e.g ketosis, hypocalcaemia and metritis.  Displacement to the left results in a reduced flow of ingesta as well as reduced digestion resulting in anorexia and [[Dehydration - Pathology|dehydration]].  

Abomasal atony and increased gas production leads to displacement. Factors reducing abomasal motility include a high concentrate diet, increased [[Volatile Fatty Acids - Anatomy & Physioogy|volatile fatty acids]] from the rumen and periparturient disease e.g ketosis, hypocalcaemia and metritis.  Displacement to the left results in a reduced flow of ingesta as well as reduced digestion resulting in anorexia and [[Dehydration - Pathology|dehydration]].