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→Extracellular Infection
==Extracellular Infection==
==Extracellular Infection==
The response to extracellular infection involves [[Complement|complement]] and phagocytosis; [[Lymphocytes#B Cells|B cell]] and T helper type II cell stimulation and the production of [[Immunoglobulin M|IgM]], which activates the classical cascade. There is also class switching of [[Immunoglobulin M|IgM]] to [[Immunoglobulin G|IgG]], which is a good opsonin and targets bacterial Fcγ receptor expressed by [[Macrophages|macrophages]] and [[Neutrophils|neutrophils]].
The response to extracellular infection involves [[Complement|complement]] and phagocytosis; [[Lymphocytes#B Cells|B cell]] and T<sub>H</sub>2 cell stimulation and the production of [[Immunoglobulin M|IgM]], which activates the classical cascade. T<sub>H</sub>17 stimulation also enhances extravasation of neutrophils to the area to clear the pathogen. There is also class switching of [[Immunoglobulin M|IgM]] to [[Immunoglobulin G|IgG]], which is a good opsonin and targets bacterial Fcγ receptor expressed by [[Macrophages|macrophages]] and [[Neutrophils|neutrophils]].
==Vesicular Infection==
==Vesicular Infection==