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− | |backcolour = FAFAD2
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− | |linkpage =Endocrine System - Pathology
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− | |linktext =Endocrine System
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− | |maplink = Endocrine System (Content Map) - Pathology
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− | |pagetype =Pathology
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− | |sublink1=Pituitary Gland - Pathology
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− | |subtext1=PITUITARY GLAND
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| Clinical signs due to a hypothalamic dysfunction. | | Clinical signs due to a hypothalamic dysfunction. |
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− | Decresaed synthesis and release of GnRH means that the pituitary is not stimulated to release FSH or LH at normal levels. Clincally seen as:
| + | Decreased synthesis and release of GnRH means that the pituitary is not stimulated to release FSH or LH at normal levels. Clinically seen as: |
| *Small, atrophic genitals. | | *Small, atrophic genitals. |
| *Feminisation in the male due to lower testosterone release. | | *Feminisation in the male due to lower testosterone release. |
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− | Hypothalamus damage also allows incresaed appetite and the animal will be: | + | Hypothalamus damage also allows increased appetite and the animal may be obese, with excess deposition of subcutaneous fat. |
− | *Obese.
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− | *Excess deposition of sub-cut fat.
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| Compression of local structures also leads to: | | Compression of local structures also leads to: |
− | *Mental retartdation. | + | *Mental retardation. |
| *Diabetes insipidus. | | *Diabetes insipidus. |
| *Visual impairment. | | *Visual impairment. |
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| + | [[Category:To Do - Reproductive]] |
| + | [[Category:Hypothalamus - Pathology]] |
| + | [[Category:Reproductive System - Pathology]] |