Difference between revisions of "Oesophageal Diverticula"

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A circumscribed sacculation of the oesophageal wall. These can be divided into congenital or acquired. Congenital diverticula are due to a herniation of the mucosa through a muscularis defect. Acquired diverticula can be subdivided into:
 
A circumscribed sacculation of the oesophageal wall. These can be divided into congenital or acquired. Congenital diverticula are due to a herniation of the mucosa through a muscularis defect. Acquired diverticula can be subdivided into:
 
#'''Traction forms''' consists of all the oesophageal layers and results from peri-oesophageal inflammation and fibrosis and develop mainly in the cranial and mid-oesophageal body. Sacculations are created by adhesions to adjacent tissues.
 
#'''Traction forms''' consists of all the oesophageal layers and results from peri-oesophageal inflammation and fibrosis and develop mainly in the cranial and mid-oesophageal body. Sacculations are created by adhesions to adjacent tissues.
#'''Pulsion forms''' includes the epitheliuma nd connective tissue and results from:
+
#'''Pulsion forms''' includes the epithelium and connective tissue and results from:
 
*an increase in intraluminal oesophageal pressure
 
*an increase in intraluminal oesophageal pressure
 
*abnormal local oesophageal motiltiy
 
*abnormal local oesophageal motiltiy

Revision as of 11:15, 10 August 2009



Typical Signalment

Description

A circumscribed sacculation of the oesophageal wall. These can be divided into congenital or acquired. Congenital diverticula are due to a herniation of the mucosa through a muscularis defect. Acquired diverticula can be subdivided into:

  1. Traction forms consists of all the oesophageal layers and results from peri-oesophageal inflammation and fibrosis and develop mainly in the cranial and mid-oesophageal body. Sacculations are created by adhesions to adjacent tissues.
  2. Pulsion forms includes the epithelium and connective tissue and results from:
  • an increase in intraluminal oesophageal pressure
  • abnormal local oesophageal motiltiy
  • deep oesophageal inflammation
  • a stenosis interupting normal peristalsis
  • vascular ring abnormalities
  • lodged foreign bodies

Diagnosis

Clinical Signs

usually result from food/fluid impaction in the sacculated area and can present as:

  • regurgitation
  • odynophagia
  • retching
  • postprandial dyspnoea
  • anorexia
  • sepsis if the diverticulum becomes perforated
  • may be subclinical if small

Laboratory Tests

Diagnostic Imaging

Treatment

Prognosis

References