Difference between revisions of "Oesophageal Diverticula"

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==Typical Signalment==
 
==Typical Signalment==
 
+
*Rare in small animals
 +
*More common in dogs than cats
 +
*No important breed or sex predisposition
 
==Description==  
 
==Description==  
 
A circumscribed sacculation of the oesophageal wall. These can be divided into congenital or acquired. Congenital diverticula are due to a herniation of the mucosa through a muscularis defect. Acquired diverticula can be subdivided into:
 
A circumscribed sacculation of the oesophageal wall. These can be divided into congenital or acquired. Congenital diverticula are due to a herniation of the mucosa through a muscularis defect. Acquired diverticula can be subdivided into:
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*may be subclinical if small
 
*may be subclinical if small
 
===Laboratory Tests===
 
===Laboratory Tests===
 
+
Normal
 
===Diagnostic Imaging===
 
===Diagnostic Imaging===
 
+
'''Survey radiography''': may show an air/soft tissue density next to or involving the oesophagus
 +
'''Contrast radiography''': will show focal dilation or a partial or complete filling of contrast media in the lumen of the oesophagus.
 +
'''Endoscopy''': will confirm the diagnosis
 
==Treatment==
 
==Treatment==
  

Revision as of 11:33, 10 August 2009



Typical Signalment

  • Rare in small animals
  • More common in dogs than cats
  • No important breed or sex predisposition

Description

A circumscribed sacculation of the oesophageal wall. These can be divided into congenital or acquired. Congenital diverticula are due to a herniation of the mucosa through a muscularis defect. Acquired diverticula can be subdivided into:

  1. Traction forms consists of all the oesophageal layers and results from peri-oesophageal inflammation and fibrosis and develop mainly in the cranial and mid-oesophageal body. Sacculations are created by adhesions to adjacent tissues.
  2. Pulsion forms includes the epithelium and connective tissue and results from:
  • an increase in intraluminal oesophageal pressure
  • abnormal local oesophageal motiltiy
  • deep oesophageal inflammation
  • a stenosis interupting normal peristalsis
  • vascular ring abnormalities
  • lodged foreign bodies

Diagnosis

Clinical Signs

usually result from food/fluid impaction in the sacculated area and can present as:

  • regurgitation
  • odynophagia
  • retching
  • postprandial dyspnoea
  • anorexia
  • sepsis if the diverticulum becomes perforated
  • may be subclinical if small

Laboratory Tests

Normal

Diagnostic Imaging

Survey radiography: may show an air/soft tissue density next to or involving the oesophagus Contrast radiography: will show focal dilation or a partial or complete filling of contrast media in the lumen of the oesophagus. Endoscopy: will confirm the diagnosis

Treatment

Prognosis

References