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| ==Treatment== | | ==Treatment== |
− | ===Primary lymphangiectasia===
| + | *Identify and treat the underlying cause if it is caused by secondary lymphangiectasia |
| *Supplementation of fat soluble vitamins | | *Supplementation of fat soluble vitamins |
| *Anecdotal report of glutamine supplementation | | *Anecdotal report of glutamine supplementation |
− | *Glucocorticoid e.g. Prednisolone at 1-2 mg/kg PO | + | *Prednisolone at 0.5-1.0 mg/kg PO BID |
| **Anti-inflammatory and immunosuppressive effect may be beneficial | | **Anti-inflammatory and immunosuppressive effect may be beneficial |
| **This is particularly true if there is associated lymphangitis, lipogranulomas and a lymphocytic-plasmacytic infiltration of the lamina propria. | | **This is particularly true if there is associated lymphangitis, lipogranulomas and a lymphocytic-plasmacytic infiltration of the lamina propria. |
− | | + | *Antimicrobials such as metronidazole or tylosin |
− | | + | **This may be beneficial due to their potential immunomodulatory effect and modulation of enteric flora |
− | ===Secondary lymphangiectasia===
| + | *Diuretics, preferably combinations, such as frusemide and spironolactone are used to treat effusions. |
− | *Identify and treat the underlying cause. | + | *Short term treatment with plasma or colloids can be given for plasma expandsion. |
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| ==Prognosis== | | ==Prognosis== |
− | | + | Guarded. The response to treatment is generally poor although some dogs may do well. Dogs in may be in remission for several years but the disease eventually progress to fulminant hypoproteinaemia. |
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