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| ==Prognosis== | | ==Prognosis== |
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− | Fever and muscle hyperaesthesia commonly begin to
| + | Within two to three days of clindamycin or trimethoprim-sulphonamide administration, pyrexia and many other acute signs should begin to resolve. Animals with these clinical signs have a good prognosis. If a response is not seen within this time period, an alternative anti-''Toxoplasma'' drug should be considered. However, anti-''Toxoplasma'' drugs are unlikely to completely eradicate the organism from the host, and so recurrences are common. |
− | respond within the first two to three days of clindamycin
| + | |
− | or trimethoprim-sulphonamide administration; ocular | + | Ocular and CNS toxoplasmosis respond more slowly to therapy and carry a worse prognosis. Some neuromuscular signs may be persistent due to permanent nervous damage. |
− | and CNS toxoplasmosis respond more slowly to therapy. | + | |
− | If fever or muscle hyperaesthesia are not lessening after | + | Animals with hepatic or pulmonary disease have a poor prognosis. This form of disease is generally seen in immunocompromised animals. |
− | three days of treatment, an alternate anti-Toxoplasma
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− | drug should be administered or other causes for the clinical | |
− | syndrome considered. It is unlikely that currently
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− | available anti-Toxoplasma drugs can completely eliminate
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− | the organism and so recurrences are common - | |
− | particularly in cats treated for less than four weeks.
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− | Cats with fever or muscle hyperaesthesia generally
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− | have a good prognosis. Cats with ocular or CNS toxoplasmosis
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− | have a guarded prognosis. The prognosis is poor for
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− | cats with hepatic or pulmonary disease caused by organism
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− | replication, particularly immunocompromised cats.
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| ==Links== | | ==Links== |