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==Pathology==
 
==Pathology==
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Gross necropsy lesions are minimal in the uncomplicated respiratory form of PRRS, but interstitial pneumonitis is a consistent histopathologic finding. There are no gross or histopathologjc lesions noted in aborted or stillborn fetuses.
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Gross necropsy lesions are minimal in the uncomplicated respiratory form of PRRS. Microscopically, interstitial pneumonia is the typical finding. There is a mononuclear infiltration of the alveolar walls with type 2 pneumonocyte proliferation, and necrotic debris and macrophages accumulate in the alveolar spaces. In lymph nodes, follicular hyperplasia and necrosis may be seen. Non-suppurative meningoencephalitis and choroiditis is commonly present, but severity is highly variable.
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Microscopic lesions of the fetus
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There are no gross lesions noted in aborted or stillborn fetuses, but histopathology inconsistently interstitial pneumonia, myocarditis anr pulmonary arteritis. However, these changes are not specific or diagnostic for PRRS.
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and placenta include umbilical vasculitis, interstitial
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pneumonia, myocarditis and pulmonary arteritis but are
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inconsistent and not diagnostic for PRRS (Lager and
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Ackermann, 1994; Lager and Halbur, 1996; Rossow et
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al., 1996b). There may be residual effects of PRRS
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virus infection in sows manifest as a reduced conception
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rate (Lowe et al., 2006).
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Microscopically, interstitial pneumonia
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with mononuclear septal infiltration, type 2 pneumonocyte
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proliferation and accumulations of necrotic
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debris and macrophages in alveolar spaces is evident.
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Follicular hyperplasia and necrosis may be present in
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lymph nodes. Nonsuppurative meningoencephalitis and
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choroiditis is commonly present, with highly variable
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severity
      
==Control==
 
==Control==
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