Difference between revisions of "Frohlich's Adiposogenital Syndrome"
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(New page: {{toplink |backcolour = FAFAD2 |linkpage =Endocrine System - Pathology |linktext =Endocrine System |maplink = Endocrine System (Content Map) - Pathology |pagetype =Pathology |sublink1=Pitu...) |
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Clinical signs due to a hypothalamic dysfunction. | Clinical signs due to a hypothalamic dysfunction. | ||
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*Diabetes insipidus. | *Diabetes insipidus. | ||
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Revision as of 13:51, 21 February 2011
Clinical signs due to a hypothalamic dysfunction.
Decresaed synthesis and release of GnRH means that the pituitary is not stimulated to release FSH or LH at normal levels. Clincally seen as:
- Small, atrophic genitals.
- Feminisation in the male due to lower testosterone release.
Hypothalamus damage also allows incresaed appetite and the animal will be:
- Obese.
- Excess deposition of sub-cut fat.
Compression of local structures also leads to:
- Mental retartdation.
- Diabetes insipidus.
- Visual impairment.