Difference between revisions of "Skin Environmental - Pathology"
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(Redirected page to Category:Integumentary System - Environmental Pathology) |
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− | + | ==Chemical damage== | |
+ | ===[[Contact Dermatitis]]=== | ||
+ | |||
+ | ===[[Ergot Poisoning]]=== | ||
+ | |||
+ | |||
+ | |||
+ | ===[[Fescue Poisoning]]=== | ||
+ | |||
+ | |||
+ | ===[[Selenium Poisoning]]=== | ||
+ | |||
+ | |||
+ | ==Physical damage== | ||
+ | |||
+ | ===[[Acral Lick Dermatitis]]=== | ||
+ | |||
+ | ===[[Callus]]=== | ||
+ | |||
+ | ===[[Feline Psychogenic Alopecia]]=== | ||
+ | |||
+ | |||
+ | |||
+ | ===[[Injection Site Reaction]]=== | ||
+ | |||
+ | |||
+ | ===[[Intertrigo]]=== | ||
+ | |||
+ | |||
+ | ===[[Pyotraumatic Dermatitis]]=== | ||
+ | |||
+ | |||
+ | |||
+ | ===[[Radiation Damage]]=== | ||
+ | |||
+ | |||
+ | |||
+ | ===[[Low Temperature Damage]]=== | ||
+ | |||
+ | |||
+ | |||
+ | ===[[High Temperature Damage]]== | ||
+ | |||
+ | |||
+ | ==Sunlight damage== | ||
+ | *Transient erythema may develop into sunburn erythema (warmth, swelling, pain) | ||
+ | *Diffusion of inflammatory mediators (''e.g.'' cytokines) from damaged keratinocytes and endothelial cells | ||
+ | *Photooxidation of existing melanin -> pigment darkening | ||
+ | *Melanogenesis | ||
+ | *Immune responses of skin are reduced by UV light | ||
+ | |||
+ | |||
+ | [[Category:Integumentary System - Sunlight Damage]] | ||
+ | |||
+ | |||
+ | ===[[Solar Dermatosis and Neoplasia]]=== | ||
+ | |||
+ | |||
+ | ===[[Solar Dermatitis]]=== | ||
+ | |||
+ | ===[[Photosensitisation]]=== | ||
+ | |||
+ | ===Photoenhanced dermatoses=== | ||
+ | *Many [[Skin Immunologic - Pathology|immune-mediated]] cutaneous disease are made worse by sunlight | ||
+ | **Lupus erythematosus | ||
+ | **Dermatomyositis | ||
+ | **Pemphigus erythematosus | ||
+ | *Vasculitis in extremities, especially white-haired horses | ||
+ | *Grossly: | ||
+ | **Erythematous, well circumscribed crusted lesions or hyperkeratotic [[Skin Glossary - Pathology|plaques]] | ||
+ | *Microscopically: | ||
+ | **Vasculitis of superficial dermal vessels | ||
+ | **Thrombi may be seen | ||
+ | |||
+ | [[Category:Integumentary System - Sunlight Damage]] |
Revision as of 16:12, 21 February 2011
Chemical damage
Contact Dermatitis
Ergot Poisoning
Fescue Poisoning
Selenium Poisoning
Physical damage
Acral Lick Dermatitis
Callus
Feline Psychogenic Alopecia
Injection Site Reaction
Intertrigo
Pyotraumatic Dermatitis
Radiation Damage
Low Temperature Damage
=High Temperature Damage
Sunlight damage
- Transient erythema may develop into sunburn erythema (warmth, swelling, pain)
- Diffusion of inflammatory mediators (e.g. cytokines) from damaged keratinocytes and endothelial cells
- Photooxidation of existing melanin -> pigment darkening
- Melanogenesis
- Immune responses of skin are reduced by UV light
Solar Dermatosis and Neoplasia
Solar Dermatitis
Photosensitisation
Photoenhanced dermatoses
- Many immune-mediated cutaneous disease are made worse by sunlight
- Lupus erythematosus
- Dermatomyositis
- Pemphigus erythematosus
- Vasculitis in extremities, especially white-haired horses
- Grossly:
- Erythematous, well circumscribed crusted lesions or hyperkeratotic plaques
- Microscopically:
- Vasculitis of superficial dermal vessels
- Thrombi may be seen