Difference between revisions of "Frohlich's Adiposogenital Syndrome"
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Clinical signs due to a hypothalamic dysfunction. | Clinical signs due to a hypothalamic dysfunction. | ||
| − | + | Decresaed synthesis and release of GnRH means that the pituitary is not stimulated to release FSH or LH at normal levels. Clincally seen as: | |
*Small, atrophic genitals. | *Small, atrophic genitals. | ||
*Feminisation in the male due to lower testosterone release. | *Feminisation in the male due to lower testosterone release. | ||
| − | Hypothalamus damage also allows | + | Hypothalamus damage also allows incresaed appetite and the animal will be: |
| + | *Obese. | ||
| + | *Excess deposition of sub-cut fat. | ||
Compression of local structures also leads to: | Compression of local structures also leads to: | ||
| − | *Mental | + | *Mental retartdation. |
*Diabetes insipidus. | *Diabetes insipidus. | ||
*Visual impairment. | *Visual impairment. | ||
Revision as of 18:48, 12 April 2011
Clinical signs due to a hypothalamic dysfunction.
Decresaed synthesis and release of GnRH means that the pituitary is not stimulated to release FSH or LH at normal levels. Clincally seen as:
- Small, atrophic genitals.
- Feminisation in the male due to lower testosterone release.
Hypothalamus damage also allows incresaed appetite and the animal will be:
- Obese.
- Excess deposition of sub-cut fat.
Compression of local structures also leads to:
- Mental retartdation.
- Diabetes insipidus.
- Visual impairment.