Difference between revisions of "Frohlich's Adiposogenital Syndrome"
Jump to navigation
Jump to search
Line 15: | Line 15: | ||
+ | |||
+ | [[Category:To Do - Reproductive]] | ||
[[Category:Hypothalamus - Pathology]] | [[Category:Hypothalamus - Pathology]] | ||
[[Category:Reproductive System - Pathology]] | [[Category:Reproductive System - Pathology]] |
Revision as of 18:48, 12 April 2011
Clinical signs due to a hypothalamic dysfunction.
Decresaed synthesis and release of GnRH means that the pituitary is not stimulated to release FSH or LH at normal levels. Clincally seen as:
- Small, atrophic genitals.
- Feminisation in the male due to lower testosterone release.
Hypothalamus damage also allows incresaed appetite and the animal will be:
- Obese.
- Excess deposition of sub-cut fat.
Compression of local structures also leads to:
- Mental retartdation.
- Diabetes insipidus.
- Visual impairment.