Difference between revisions of "Equine Internal Medicine Q&A 09"

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Neonatal isoerythrolysis.
 
Neonatal isoerythrolysis.
|l1=Neonatal Isoerythrolysis#Equine neonatal isoerythrolysis
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|q2=What are the three potential clinical entities that could lead to the death of this foal?
 
|q2=What are the three potential clinical entities that could lead to the death of this foal?
 
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*Hypovolaemic shock secondary to the rapid lysis of red blood cells and their functional masses as vascular expanders. <br><br>
 
*Hypovolaemic shock secondary to the rapid lysis of red blood cells and their functional masses as vascular expanders. <br><br>
 
*Loss of oxygen carrying capacity from the lysed red blood cells. <br><br>
 
*Loss of oxygen carrying capacity from the lysed red blood cells. <br><br>
*The development of kernicterus. The increasingly elevated levels of bilirubin within the bloodstream can cross the immature blood–brain barrier in the foal and result in chemical toxicity to the brain. The brain damage can be permanent and be represented by persistent seizures or maladjustment.
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*The development of kernicterus. The increasingly elevated levels of bilirubin with in the bloodstream can cross the immature blood–brain barrier in the foal and result in chemical toxicity to the brain. The brain damage can be permanent and be represented by persistent seizures or maladjustment.
|l2=Neonatal Isoerythrolysis#Clinical Signs
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Revision as of 17:57, 31 May 2011


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A 12-hour-old foal is presented as weak and lethargic. The heart rate is 96bpm, and the mucous membranes are pale and jaundiced. Laboratory evaluation reveals a haematocrit of 10%, a haemoglobin concentration of 34g/l and a total protein of 52g/l.


Question Answer Article
What is the most likely diagnosis? [[|Link to Article]]
What are the three potential clinical entities that could lead to the death of this foal? [[ |Link to Article]]


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