Difference between revisions of "Avian Encephalomyelitis Flashcards"
Jump to navigation
Jump to search
| (14 intermediate revisions by 3 users not shown) | |||
| Line 1: | Line 1: | ||
| − | <FlashCard questions=" | + | <FlashCard questions="12"> |
|q1=Name the virus that causes avian encephalomyeltits (AE)? | |q1=Name the virus that causes avian encephalomyeltits (AE)? | ||
|a1=Avian encephalomyelitis-like virus 1 (AEV). | |a1=Avian encephalomyelitis-like virus 1 (AEV). | ||
|l1=Avian Encephalomyelitis#Introduction | |l1=Avian Encephalomyelitis#Introduction | ||
| − | |q2 | + | |q2= What are the main economic concerns for breeders and layers about AE? |
| − | + | |a2= | |
| − | |||
| − | |||
| − | |||
| − | |||
| − | |||
| − | |||
| − | |||
| − | |||
| − | | | ||
*Temporary decrease in egg production in laying hens | *Temporary decrease in egg production in laying hens | ||
*Decrease in egg hatchability | *Decrease in egg hatchability | ||
*Neurological disease and morbidity /mortality of chicks under three weeks of age | *Neurological disease and morbidity /mortality of chicks under three weeks of age | ||
*Surviving chicks are considered unlikely to be profitable | *Surviving chicks are considered unlikely to be profitable | ||
| − | | | + | |l2=Avian Encephalomyelitis#Introduction |
| − | | | + | |q3= How is AE spread? |
| − | | | + | |a3= |
*Horizontally via faecal-oral route (main route of infection) | *Horizontally via faecal-oral route (main route of infection) | ||
*Vertical transmission from infected layer to their chicks | *Vertical transmission from infected layer to their chicks | ||
| − | + | |l3= Avian Encephalomyelitis#Introduction | |
| − | | | + | |q4= What species of birds does AE affect? |
| − | | | + | |a4= |
| − | | | ||
*Chickens (main domestic host) | *Chickens (main domestic host) | ||
*Species of partridge | *Species of partridge | ||
| Line 33: | Line 23: | ||
*Guineafowl | *Guineafowl | ||
*Pheasants | *Pheasants | ||
| − | | | + | |l4= Avian Encephalomyelitis#Signalment |
| − | | | + | |q5=What are the clinical signs of AE? |
| − | | | + | |a5= AE causes the most serious disease in chicks under 3 weeks of age. The clinical signs for this age group are: |
*neurological signs ranging from ataxia, rapid tremor of the head and neck, drooping of the wings, weakness, paralysis, exercise intolerance to blindness and changes in vocalisation. | *neurological signs ranging from ataxia, rapid tremor of the head and neck, drooping of the wings, weakness, paralysis, exercise intolerance to blindness and changes in vocalisation. | ||
*Weight loss, lameness, and unthriftiness | *Weight loss, lameness, and unthriftiness | ||
| Line 44: | Line 34: | ||
*Decrease in droppings | *Decrease in droppings | ||
*Decrease in hatching rates | *Decrease in hatching rates | ||
| − | | | + | |l5= Avian Encephalomyelitis#Clinical Signs |
| − | | | + | |q6= How can AE be diagnosed? |
| − | | | + | |a6= |
The following methods have been used to help diagnose Avian Encephalomyelitis (AE): | The following methods have been used to help diagnose Avian Encephalomyelitis (AE): | ||
*Virus Neutralization test | *Virus Neutralization test | ||
| Line 53: | Line 43: | ||
*Elisa test | *Elisa test | ||
All of the above tests indicate exposure to AE but not neccessarily a current infection. | All of the above tests indicate exposure to AE but not neccessarily a current infection. | ||
| − | |||
| − | | | + | Diagnosis can be achieved confirmed by either histopathology or by virus isolation. |
| − | | | + | |
| − | | | + | Post mortem and identification of pathognomonic lesions in the: |
| − | | | + | *Midbrain and rotundua and ovidalis nucleus (microglisosis |
| − | | | + | *Proventriculus (dense nodules within the muscular wall) |
| − | | | + | *Lesions can also be present in the pancreas |
| − | | | + | |l6= Avian Encephalomyelitis#Diagnosis |
| − | | | + | |q7=What is the distribution of AE? |
| − | | | + | |a7=Worldwide, the disease has been documented in Africa, Asia, Australia, Europe, and North and South America. |
| − | | | + | |l7=Avian Encephalomyelitis#Distribution |
| − | | | + | |q8=What is the treatment for AE? |
| − | | | + | |a8=There is no treatment for chicks infected with avian encephalomyelitis (AE). |
| − | | | + | |l8=Avian Encephalomyelitis#Treatment |
| − | | | + | |q9=Can chicken and surviving chicks become immune to AEV? |
| − | | | + | |a9=Yes, surviving chicks, infected hens and birds that have been vaccinated will be immune to AE for life. |
| − | | | + | |l9=Avian Encephalomyelitis#Treatment |
| − | + | |q10=How many serotypes are there for AEV? | |
| + | |a10=There is only one serotype for the AEV. | ||
| + | |l10=Avian Encephalomyelitis#Control | ||
| + | |q11=How can AE be controlled? | ||
| + | |a11=Vaccination of pullets several weeks before they come into lay. Vaccination protects the flock from a reduction in egg production and prevents vertical transmission of the virus to chicks by providing the chicks with a sufficient level of maternal derived antibodies. There are live and attenuated vaccinations available which can be administered in drinking water or as an eye drop, the latter being more effective. | ||
| + | |l11=Avian Encephalomyelitis#Control | ||
| + | ==Further links== | ||
| + | [[Avian encephalomyelitis-like virus 1| ''Avian encephalomyelitis-like virus 1'']] | ||
| − | [[Category:Avian Flashcards]] | + | </FlashCard> |
| − | [[Category:CABI | + | [[Category: Avian Flashcards]] |
| − | + | [[Category:To Do - CABI review]] | |
Revision as of 07:58, 23 June 2011
| Question | Answer | Article | |
| Name the virus that causes avian encephalomyeltits (AE)? | Avian encephalomyelitis-like virus 1 (AEV).
|
Link to Article | |
| What are the main economic concerns for breeders and layers about AE? |
|
Link to Article | |
| How is AE spread? |
|
Link to Article | |
| What species of birds does AE affect? |
|
Link to Article | |
| What are the clinical signs of AE? | AE causes the most serious disease in chicks under 3 weeks of age. The clinical signs for this age group are:
Laying hens rarely show clinical signs but the virus causes:
|
Link to Article | |
| How can AE be diagnosed? | The following methods have been used to help diagnose Avian Encephalomyelitis (AE):
All of the above tests indicate exposure to AE but not neccessarily a current infection. Diagnosis can be achieved confirmed by either histopathology or by virus isolation. Post mortem and identification of pathognomonic lesions in the:
|
Link to Article | |
| What is the distribution of AE? | Worldwide, the disease has been documented in Africa, Asia, Australia, Europe, and North and South America.
|
Link to Article | |
| What is the treatment for AE? | There is no treatment for chicks infected with avian encephalomyelitis (AE).
|
Link to Article | |
| Can chicken and surviving chicks become immune to AEV? | Yes, surviving chicks, infected hens and birds that have been vaccinated will be immune to AE for life.
|
Link to Article | |
| How many serotypes are there for AEV? | There is only one serotype for the AEV.
|
Link to Article | |
| How can AE be controlled? | Vaccination of pullets several weeks before they come into lay. Vaccination protects the flock from a reduction in egg production and prevents vertical transmission of the virus to chicks by providing the chicks with a sufficient level of maternal derived antibodies. There are live and attenuated vaccinations available which can be administered in drinking water or as an eye drop, the latter being more effective.
|
[[Avian Encephalomyelitis#Control |Link to Article]] | |
| [[|Link to Article]] | |||