Difference between revisions of "Reptiles and Amphibians Q&A 12"
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Metabolic bone disease. | Metabolic bone disease. | ||
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|q2=What poor techniques of captive management might be the cause of this turtle’s disorder? | |q2=What poor techniques of captive management might be the cause of this turtle’s disorder? | ||
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A diet rich in phosphorus and low in calcium; hypovitaminosis D3; and lack of exposure to unfiltered sunlight or full-spectrum ultraviolet light can cause metabolic bone disease. | A diet rich in phosphorus and low in calcium; hypovitaminosis D3; and lack of exposure to unfiltered sunlight or full-spectrum ultraviolet light can cause metabolic bone disease. | ||
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|q3=How is this condition treated? | |q3=How is this condition treated? | ||
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Although the bony carapace and plastron will eventually become remineralised when the turtle’s diet is improved, the deformities are likely to remain apparent for the life of the turtle. | Although the bony carapace and plastron will eventually become remineralised when the turtle’s diet is improved, the deformities are likely to remain apparent for the life of the turtle. | ||
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</FlashCard> | </FlashCard> | ||
Revision as of 18:06, 7 September 2011
These two three-yearold female red-eared slider turtles are being kept under different captive husbandry conditions. The turtle on the right is normal.
| Question | Answer | Article | |
| What is your diagnosis of the turtle on the left? | Metabolic bone disease. |
[[|Link to Article]] | |
| What poor techniques of captive management might be the cause of this turtle’s disorder? | A diet rich in phosphorus and low in calcium; hypovitaminosis D3; and lack of exposure to unfiltered sunlight or full-spectrum ultraviolet light can cause metabolic bone disease. |
[[|Link to Article]] | |
| How is this condition treated? | Change to a more natural high-calcium, moderate-phosphorus diet that contains bony tissue, eg, live fish or commercial floating turtle chow, and calcium-rich leafy vegetation and algae. If deemed appropriate, calcium lactate or gluconate can be administered orally or parenterally (1.0–2.5mg/kg daily). Although the bony carapace and plastron will eventually become remineralised when the turtle’s diet is improved, the deformities are likely to remain apparent for the life of the turtle. |
[[|Link to Article]] | |
