Difference between revisions of "Placenta Endocrine Function - Anatomy & Physiology"
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+ | {{toplink | ||
+ | |backcolour = FAFAD2 | ||
+ | |linkpage =Endocrine System - Anatomy & Physiology | ||
+ | |linktext =Endocrine System | ||
+ | |maplink = Endocrine System (Content Map) - Anatomy & Physiology | ||
+ | |pagetype =Anatomy | ||
+ | |subtext1=REPRODUCTIVE SYSTEM | ||
+ | |sublink1=Reproductive System | ||
+ | }} | ||
+ | <br> | ||
− | The Placenta is a hugely important endocrine organ, producing many [[Hormones - Anatomy & Physiology|hormones]] which affect the status of pregnancy and the maternal physiology. | + | |
+ | The Placenta is a hugely important endocrine organ, producing many [[Endocrine System - Hormones - Anatomy & Physiology|hormones]] which affect the status of pregnancy and the maternal physiology. | ||
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!Action | !Action | ||
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− | |Prostaglandin F2a | + | |<font color=Purple>Prostaglandin F2a</font> |
|Produced towards the end of pregnancy in increasing amounts to allow for parturition. | |Produced towards the end of pregnancy in increasing amounts to allow for parturition. | ||
|Causes luteolysis and promotes uterine contractions. | |Causes luteolysis and promotes uterine contractions. | ||
|- | |- | ||
− | |Progesterone | + | |<font color=Purple>Progesterone</font> |
|The Placenta takes over Progesterone production from the Corpus Luteum to maintain the pregnancy. | |The Placenta takes over Progesterone production from the Corpus Luteum to maintain the pregnancy. | ||
|Maintains the pregnancy by thickening the cervix, depressing the maternal immune response and preventing ovulation. It also promotes mammary growth. | |Maintains the pregnancy by thickening the cervix, depressing the maternal immune response and preventing ovulation. It also promotes mammary growth. | ||
|- | |- | ||
− | |Oestrogen | + | |<font color=Purple>Oestrogen<font> |
|Oestrogen levels rise throughout pregnancy as the placenta grows. | |Oestrogen levels rise throughout pregnancy as the placenta grows. | ||
− | |Stimulate the growth of the myometrium, promote mammary development and suppress gonadotrophin release from the [[Pituitary Gland - Anatomy & Physiology|pituitary gland]]. | + | |Stimulate the growth of the myometrium, promote mammary development and suppress gonadotrophin release from the [[Endocrine System - Pituitary Gland - Anatomy & Physiology|pituitary gland]]. |
|- | |- | ||
− | |Placental Lactogen | + | |<font color=Purple>Placental Lactogen<font> |
− | |This is related to [[Lactation - Endocrine Control - Anatomy & Physiology|Prolactin]] and [[Pituitary Growth Hormone - Anatomy & Physiology|Growth hormone]], and is produced by the placenta and uterus. | + | |This is related to [[Lactation - Endocrine Control - Anatomy & Physiology|Prolactin]] and [[Endocrine System - Pituitary Growth Hormone - Anatomy & Physiology|Growth hormone]], and is produced by the placenta and uterus. |
|Promotes growth and differentiation of mammary tissue. Identified in primates, ruminants and rodents. | |Promotes growth and differentiation of mammary tissue. Identified in primates, ruminants and rodents. | ||
|- | |- | ||
− | |Equine Chorionic Gonadotropin (eCG) | + | |<font color=Purple>Equine Chorionic Gonadotropin (eCG)<font> |
|This is produced by foetal trophoblast cells, and is actually the same molecule as equine LH. | |This is produced by foetal trophoblast cells, and is actually the same molecule as equine LH. | ||
− | |Prevents luteal regression and is thus the signal for [[Maternal Recognition of Pregnancy - Anatomy & Physiology|Maternal Recognition of Pregnancy]] in equids. | + | |Prevents luteal regression and is thus the signal for [[Female Reproductive Tract -Maternal Recognition of Pregnancy - Anatomy & Physiology|Maternal Recognition of Pregnancy]] in equids. |
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− | |Human Chorionic Gonadotropin (hCG) | + | |<font color=Purple>Human Chorionic Gonadotropin (hCG)<font> |
|This is produced by foetal trophoblast cells. | |This is produced by foetal trophoblast cells. | ||
− | |Prevents luteal regression and is thus the signal for [[Maternal Recognition of Pregnancy - Anatomy & Physiology|Maternal Recognition of Pregnancy]] in primates. | + | |Prevents luteal regression and is thus the signal for [[Female Reproductive Tract -Maternal Recognition of Pregnancy - Anatomy & Physiology|Maternal Recognition of Pregnancy]] in primates. |
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Revision as of 21:56, 4 August 2008
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The Placenta is a hugely important endocrine organ, producing many hormones which affect the status of pregnancy and the maternal physiology.
Hormone | Production and Regulation | Action |
---|---|---|
Prostaglandin F2a | Produced towards the end of pregnancy in increasing amounts to allow for parturition. | Causes luteolysis and promotes uterine contractions. |
Progesterone | The Placenta takes over Progesterone production from the Corpus Luteum to maintain the pregnancy. | Maintains the pregnancy by thickening the cervix, depressing the maternal immune response and preventing ovulation. It also promotes mammary growth. |
Oestrogen | Oestrogen levels rise throughout pregnancy as the placenta grows. | Stimulate the growth of the myometrium, promote mammary development and suppress gonadotrophin release from the pituitary gland. |
Placental Lactogen | This is related to Prolactin and Growth hormone, and is produced by the placenta and uterus. | Promotes growth and differentiation of mammary tissue. Identified in primates, ruminants and rodents. |
Equine Chorionic Gonadotropin (eCG) | This is produced by foetal trophoblast cells, and is actually the same molecule as equine LH. | Prevents luteal regression and is thus the signal for Maternal Recognition of Pregnancy in equids. |
Human Chorionic Gonadotropin (hCG) | This is produced by foetal trophoblast cells. | Prevents luteal regression and is thus the signal for Maternal Recognition of Pregnancy in primates. |