Difference between revisions of "Immunity to Fungi"

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Cutaneous and superficial fungal infections are normally self-limiting, with recovery associated with a resistance to re-infection. As hosts often develop delayed-type [[Hypersensitivity - Introduction|hypersensitivity]], this resistance appears to be cell-mediated.
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Cutaneous and superficial fungal infections are normally self-limiting, with recovery associated with a resistance to re-infection. As hosts often develop delayed-type [[Hypersensitivity - WikiBlood|hypersensitivity]], this resistance appears to be cell-mediated.
*[[T cell differentiation|T cell]] immunity- it is thought that [[T cell differentiation#TH1 Cells|helper T cells]] release cytokines to activate [[Macrophages|macrophages]] to fight the infection
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*[[T cell differentiation - WikiBlood|T cell]] immunity- it is thought that [[T cell differentiation - WikiBlood#TH1 Cells|helper T cells]] release cytokines to activate [[Macrophages|macrophages]] to fight the infection
*[[Neutrophils|Neutrophils]]- there is now growing evidence for the role of [[Neutrophils|neutrophils]] in some infections of the respiratory system, with defensin and the nitric oxide pathways implicated
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*[[Neutrophils|Neutrophils]]- there is now growing evidence for the role of [[Neutrophils|neutrophils]] in some infections of the respiratory system, with defensin and the nitric oxide pathways implicated[[Category:Fungi]]
 
 
==Test yourself with the Fungi Flashcard==
 
 
 
[[Fungi_Flashcards_-_WikiBugs|Fungi Flashcards]]
 
 
 
[[Category:Fungi]]
 
 
[[Category:To_Do_-_Fungi]]
 
[[Category:To_Do_-_Fungi]]

Revision as of 11:32, 25 June 2010

Cutaneous and superficial fungal infections are normally self-limiting, with recovery associated with a resistance to re-infection. As hosts often develop delayed-type hypersensitivity, this resistance appears to be cell-mediated.

  • T cell immunity- it is thought that helper T cells release cytokines to activate macrophages to fight the infection
  • Neutrophils- there is now growing evidence for the role of neutrophils in some infections of the respiratory system, with defensin and the nitric oxide pathways implicated