Difference between revisions of "Small Intestine Impaction - Horse"
(5 intermediate revisions by the same user not shown) | |||
Line 1: | Line 1: | ||
− | {{ | + | {{Unfinished}} |
− | == | + | |
− | + | ||
− | '''Small Intestinal impaction''' causes total or partial obstruction of the intestinal lumen resulting in [[:Category:Colic in Horses|colic]]. The obstruction is most often associated with [[Ascarid Impaction - Horse|ascarid impaction]] due to ascarid infection<ref>Cribb NC, Cote NM, Bouré LP, Peregrine AS. (2006). ''Acute small intestinal obstruction associated with Parascaris equorum infection in young horses: 25 cases (1985-2004).''. New Zealand Veterinary Journal</ref> in young horses, or ileal impaction due to ingestion of Bermuda grass in the USA or | + | ==Description== |
+ | '''Small Intestinal impaction''' causes total or partial obstruction of the intestinal lumen resulting in [[:Category:Colic in Horses|colic]]. The obstruction is most often associated with [[Ascarid Impaction - Horse|ascarid impaction]] due to ascarid infection<ref>Cribb NC, Cote NM, Bouré LP, Peregrine AS. (2006). ''Acute small intestinal obstruction associated with Parascaris equorum infection in young horses: 25 cases (1985-2004).''. New Zealand Veterinary Journal</ref> in young horses, or ileal impaction due to ingestion of Bermuda grass in the USA or tapeworm infection in the UK. Ingesta and fluid builds up proximal to the impaction and does not reach the absorptive large intestine, this results in systemic volume depletion and reduced cardiac output. | ||
Impaction may also result due to postoperative ileus; the risk can be minimised by good surgical technique, appropriate postoperative care and the administration of prokinectic drugs. | Impaction may also result due to postoperative ileus; the risk can be minimised by good surgical technique, appropriate postoperative care and the administration of prokinectic drugs. | ||
Line 11: | Line 12: | ||
Ileal impaction is more common in younger animals which are at greater risk of infection with tapeworms such as ''[[Anoplocephala|Anoplocephala perfoliata]].'' | Ileal impaction is more common in younger animals which are at greater risk of infection with tapeworms such as ''[[Anoplocephala|Anoplocephala perfoliata]].'' | ||
− | ==Clinical Signs== | + | ==Diagnosis== |
+ | ===Clinical Signs=== | ||
Clinical signs are of simple small intestine obstruction. Typically the condition starts with mild [[Colic Diagnosis - Clinical Signs|signs of colic]] which then progress as the condition becomes established. There will be additional signs of parasite infestation, weight loss, diarrhoea and ill thrift, in cases associated with ascarid and tapeworm infection. | Clinical signs are of simple small intestine obstruction. Typically the condition starts with mild [[Colic Diagnosis - Clinical Signs|signs of colic]] which then progress as the condition becomes established. There will be additional signs of parasite infestation, weight loss, diarrhoea and ill thrift, in cases associated with ascarid and tapeworm infection. | ||
The severity and speed of onset of clinical signs is determined by the level of the obstruction. Proximal obstructions will progress more rapidly than distal ones. | The severity and speed of onset of clinical signs is determined by the level of the obstruction. Proximal obstructions will progress more rapidly than distal ones. | ||
− | + | '''Passage of a stomach tube''' will produce nasogastric reflux depending on the site of obstruction and its duration; for a proximal obstruction fluid will build up in the stomach after approximately 3-6 hours, and for a distal obstruction, after 12 hours. | |
'''Gut sounds''' will decrease as the small intestine becomes distended and atonic. | '''Gut sounds''' will decrease as the small intestine becomes distended and atonic. | ||
On '''[[Rectal Examination of the Horse|rectal examination]]''' the small intestine is palpable as distended loops; in the early stages of ileal impaction a tubular structure may be felt in the mid abdomen. | On '''[[Rectal Examination of the Horse|rectal examination]]''' the small intestine is palpable as distended loops; in the early stages of ileal impaction a tubular structure may be felt in the mid abdomen. | ||
− | Clinical signs and rectal examination will diagnose the problem to the small | + | Clinical signs and rectal examination will diagnose the problem to the small intesine but it may not be possible to make a definitive diagnosis until the horse is taken to surgery. |
+ | |||
See '''[[:Category:Colic Diagnosis in the Horse|Colic Diagnosis in Horses]]''' | See '''[[:Category:Colic Diagnosis in the Horse|Colic Diagnosis in Horses]]''' | ||
===Ultrasound=== | ===Ultrasound=== | ||
− | + | Abdominal ultrasound reveals distended loops of small intestine which are frequently immotile. | |
==Treatment== | ==Treatment== | ||
− | In cases of partial impaction medical treatment with laxatives, IV fluids and analgesia may be successful but surgical management is usually recommended for small intestinal impaction. A ventral midline laparotomy is carried out to gain access to the small intestine, in severe cases the impacted portion of the intestine is removed and an anastamosis performed. In mild cases the impaction may be manually reduced but the | + | In cases of partial impaction medical treatment with laxatives, IV fluids and analgesia may be successful but surgical management is usually recommended for small intestinal impaction. A ventral midline laparotomy is carried out to gain access to the small intestine, in severe cases the impacted portion of the intestine is removed and an anastamosis performed. In mild cases the impaction may be manually reduced but the intesine must be inspected closely for viability and removed if it damaged. In the case of ascarid impaction several enterotomies are usually performed. |
See '''[[Colic, Medical Treatment|Medical Treatment of Colic in Horses]]''' | See '''[[Colic, Medical Treatment|Medical Treatment of Colic in Horses]]''' | ||
Line 36: | Line 39: | ||
If the impaction causes complete obstruction to the small intestine then the prognosis is poor, with mortality rates of up to 92%. | If the impaction causes complete obstruction to the small intestine then the prognosis is poor, with mortality rates of up to 92%. | ||
− | + | ==Refernces== | |
− | |||
− | |||
− | |||
− | == | ||
− | |||
* Mair, T.S, Divers, T.J, Ducharme, N.G (2002) '''Manual of Equine Gastroenterology''', ''WB Saunders''. | * Mair, T.S, Divers, T.J, Ducharme, N.G (2002) '''Manual of Equine Gastroenterology''', ''WB Saunders''. | ||
* Merck & Co (2008) '''The Merck Veterinary Manual (Eighth Edition)''' ''Merial'' | * Merck & Co (2008) '''The Merck Veterinary Manual (Eighth Edition)''' ''Merial'' | ||
− | |||
− | |||
− | |||
[[Category:Surgical Colic in the Horse]] | [[Category:Surgical Colic in the Horse]] | ||
− | [[Category: | + | [[Category:To_Do_-_lizzyk]] |
− | [[Category: | + | [[Category:To_Do_-_Review]] |
− | |||
[[Category:Impaction - Horse]] | [[Category:Impaction - Horse]] |
Revision as of 21:59, 6 October 2010
This article is still under construction. |
Description
Small Intestinal impaction causes total or partial obstruction of the intestinal lumen resulting in colic. The obstruction is most often associated with ascarid impaction due to ascarid infection[1] in young horses, or ileal impaction due to ingestion of Bermuda grass in the USA or tapeworm infection in the UK. Ingesta and fluid builds up proximal to the impaction and does not reach the absorptive large intestine, this results in systemic volume depletion and reduced cardiac output.
Impaction may also result due to postoperative ileus; the risk can be minimised by good surgical technique, appropriate postoperative care and the administration of prokinectic drugs.
Signalment
Foals and yearlings are particularly susceptible to infection with ascarids, impaction usually occurs following the administration of an anthelmintic with a high efficacy.
Ileal impaction is more common in younger animals which are at greater risk of infection with tapeworms such as Anoplocephala perfoliata.
Diagnosis
Clinical Signs
Clinical signs are of simple small intestine obstruction. Typically the condition starts with mild signs of colic which then progress as the condition becomes established. There will be additional signs of parasite infestation, weight loss, diarrhoea and ill thrift, in cases associated with ascarid and tapeworm infection.
The severity and speed of onset of clinical signs is determined by the level of the obstruction. Proximal obstructions will progress more rapidly than distal ones.
Passage of a stomach tube will produce nasogastric reflux depending on the site of obstruction and its duration; for a proximal obstruction fluid will build up in the stomach after approximately 3-6 hours, and for a distal obstruction, after 12 hours. Gut sounds will decrease as the small intestine becomes distended and atonic.
On rectal examination the small intestine is palpable as distended loops; in the early stages of ileal impaction a tubular structure may be felt in the mid abdomen.
Clinical signs and rectal examination will diagnose the problem to the small intesine but it may not be possible to make a definitive diagnosis until the horse is taken to surgery.
Ultrasound
Abdominal ultrasound reveals distended loops of small intestine which are frequently immotile.
Treatment
In cases of partial impaction medical treatment with laxatives, IV fluids and analgesia may be successful but surgical management is usually recommended for small intestinal impaction. A ventral midline laparotomy is carried out to gain access to the small intestine, in severe cases the impacted portion of the intestine is removed and an anastamosis performed. In mild cases the impaction may be manually reduced but the intesine must be inspected closely for viability and removed if it damaged. In the case of ascarid impaction several enterotomies are usually performed.
See Medical Treatment of Colic in Horses
Prognosis
If the impaction causes complete obstruction to the small intestine then the prognosis is poor, with mortality rates of up to 92%.
Refernces
- Mair, T.S, Divers, T.J, Ducharme, N.G (2002) Manual of Equine Gastroenterology, WB Saunders.
- Merck & Co (2008) The Merck Veterinary Manual (Eighth Edition) Merial
- ↑ Cribb NC, Cote NM, Bouré LP, Peregrine AS. (2006). Acute small intestinal obstruction associated with Parascaris equorum infection in young horses: 25 cases (1985-2004).. New Zealand Veterinary Journal