Difference between revisions of "Porcine Parvovirus"
Jump to navigation
Jump to search
| Line 1: | Line 1: | ||
{{unfinished}} | {{unfinished}} | ||
| − | |||
| − | |||
| − | |||
| − | |||
| − | |||
| − | |||
| − | |||
| − | |||
| − | |||
====Antigenicity==== | ====Antigenicity==== | ||
| Line 37: | Line 28: | ||
*Inactivated vaccine (UK) or live attenuated (USA) | *Inactivated vaccine (UK) or live attenuated (USA) | ||
*Infected piglets should be culled | *Infected piglets should be culled | ||
| + | [[Category:Parvoviridae]][[Category:Pig]] | ||
Revision as of 11:21, 23 May 2010
 |
This article is still under construction. |
Antigenicity
- Single serotype
Hosts
- Pigs
Pathogenesis
- Principal viral cause of SMEDI syndrome: Stillbirths, Mummification, Embryonic Deaths, and Infertility
- Replicates in gastrointestinal epithelium followed by viremia
- Often no clinical signs in breeding and growing stock
- Transplacental infection occurs in early pregnancy, with some (but not all) surviving piglets living as carriers
Epidemiology
- About 50% of UK pigs are antibody-positive
- Transfer is orofecal or via fomites
- Outbreaks occur as herd is developing immunity
- Congenitally infected piglets act as herd reservoir
Diagnosis
- Immunostaining of fetal tissues
- Virus isolation in cell culture
- HA on feces
- Rising antibody titres in paired serum sample by HAI
Control
- Inactivated vaccine (UK) or live attenuated (USA)
- Infected piglets should be culled