Difference between revisions of "Category:Forestomach - Inflammatory Pathology"

Revision as of 12:03, 29 May 2010

Opportunistic pathogens take advantage of acidotic lesions and other disturbances of rumen flora.
- e.g. F. necrophorum, fungi
- Infection with Mucor gives mucormycosis- chronic ongoing granulomatous rumenitis.
- Also agents which cause lesions in other areas of tract e.g. BVDV, actinobacillosis.

Multiple, dark red areas of swollen papillae, mainly in ventral sac and pillars.

Coagulative necrosis of papillae
Marked neutrophil infiltrate
Serofibrinous exudate
Thromboembolic spread to form areas of coagulative necrosis and abscesses in liver.

The following 3 pages are in this category, out of 3 total.

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-===Mucormycosis===
+===[[Mucormycosis]]===
-* ''Mucor'', a fungi, invades the [[The Rumen - Anatomy & Physiology|rumen]] wall, e.g. after damage caused by [[Rumenal Acidosis|rumenal acidosis]].
-* Produces chronic ongoing granulomatous rumenitis.
-* Difficult to diagnose in life.
-* May produce pain and poor growth
-* Infective process may penetrate wall and produce peritonitis.
-====Pathology====
-=====Gross=====
-* Thickening and ulceration of mucosal surface of [[The Rumen - Anatomy & Physiology|rumen]].
-** Feels like thick leather.
-*[[The Rumen - Anatomy & Physiology|Rumen]] wall becomes fibrosed.
-=====Histological=====
-* Classical granulomatous inflammation
-** May have giant cells in addition to sheets of [[Macrophages - WikiBlood|macrophages]].
-* Can visualise fungal hyphae in tissue using special stains (PAS),
-** Often grow along blood vessels.
-*** May cause thrombosis of small blood vessels, leading to further necrosis (infarction) of [[The Rumen - Anatomy & Physiology|rumen]] wall.
 ==Neutrophilic Inflammation==