Difference between revisions of "Skin Environmental - Pathology"

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#REDIRECT[[:Category:Integumentary System - Environmental Pathology]]
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==Chemical damage==
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===[[Contact Dermatitis]]===
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===[[Ergot Poisoning]]===
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===[[Fescue Poisoning]]===
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===[[Selenium Poisoning]]===
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==Physical damage==
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===[[Acral Lick Dermatitis]]===
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===[[Callus]]===
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===[[Feline Psychogenic Alopecia]]===
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===[[Injection Site Reaction]]===
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===[[Intertrigo]]===
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===[[Pyotraumatic Dermatitis]]===
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===[[Radiation Damage]]===
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===[[Low Temperature Damage]]===
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===[[High Temperature Damage]]==
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==Sunlight damage==
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*Transient erythema may develop into sunburn erythema (warmth, swelling, pain)
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*Diffusion of inflammatory mediators (''e.g.'' cytokines) from damaged keratinocytes and endothelial cells
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*Photooxidation of existing melanin -> pigment darkening
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*Melanogenesis
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*Immune responses of skin are reduced by UV light
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[[Category:Integumentary System - Sunlight Damage]]
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===[[Solar Dermatosis and Neoplasia]]===
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===Solar dermatitis===
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*Particularly in white animals and where little or no hair is present
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*Grossly:
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**[[Skin Glossary - Pathology|Erythema, scaling and crusting]]
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**-> Wrinkled nand thickened skin
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** Squamous cell carcinoma or haemangiosarcoma/haemangioma may develop
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*Microscopically:
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**Dyskeratotic cells
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**Intercellular oedema
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**Vacuolated keratinocytes
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**Followed by [[Skin Glossary - Pathology|hyperkeratosis, parakeratosis and acanthosis]]
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**Endothelial swelling
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**Haemorrhage
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**Hyperplasia
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**Dermal fibrosis
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**Dogs may develop actinic [[Skin Glossary - Pathology|comedones]]
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[[Category:Integumentary System - Sunlight Damage]]
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===[[Photosensitisation]]===
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===Photoenhanced dermatoses===
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*Many [[Skin Immunologic - Pathology|immune-mediated]] cutaneous disease are made worse by sunlight
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**Lupus erythematosus
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**Dermatomyositis
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**Pemphigus erythematosus
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*Vasculitis in extremities, especially white-haired horses
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*Grossly:
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**Erythematous, well circumscribed crusted lesions or hyperkeratotic [[Skin Glossary - Pathology|plaques]]
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*Microscopically:
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**Vasculitis of superficial dermal vessels
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**Thrombi may be seen
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[[Category:Integumentary System - Sunlight Damage]]

Revision as of 16:12, 21 February 2011

Chemical damage

Contact Dermatitis

Ergot Poisoning

Fescue Poisoning

Selenium Poisoning

Physical damage

Acral Lick Dermatitis

Callus

Feline Psychogenic Alopecia

Injection Site Reaction

Intertrigo

Pyotraumatic Dermatitis

Radiation Damage

Low Temperature Damage

=High Temperature Damage

Sunlight damage

  • Transient erythema may develop into sunburn erythema (warmth, swelling, pain)
  • Diffusion of inflammatory mediators (e.g. cytokines) from damaged keratinocytes and endothelial cells
  • Photooxidation of existing melanin -> pigment darkening
  • Melanogenesis
  • Immune responses of skin are reduced by UV light


Solar Dermatosis and Neoplasia

Solar dermatitis

  • Particularly in white animals and where little or no hair is present
  • Grossly:
  • Microscopically:

Photosensitisation

Photoenhanced dermatoses

  • Many immune-mediated cutaneous disease are made worse by sunlight
    • Lupus erythematosus
    • Dermatomyositis
    • Pemphigus erythematosus
  • Vasculitis in extremities, especially white-haired horses
  • Grossly:
    • Erythematous, well circumscribed crusted lesions or hyperkeratotic plaques
  • Microscopically:
    • Vasculitis of superficial dermal vessels
    • Thrombi may be seen
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