Difference between revisions of "Important Hormonal Regulators of the Kidney - Renal Flash Cards - Anatomy & Physiology"
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'''Use the mouse to highlight the answers. They are written in white''' | '''Use the mouse to highlight the answers. They are written in white''' | ||
+ | |||
+ | <TABLE BORDER="2" BORDERCOLOR="#6600FF" CELLPADDING="2" CELLSPACING="2" WIDTH="80%"> | ||
+ | <TR> | ||
+ | <TD>'''Question'''</TD> | ||
+ | <TD>'''Answer'''</TD> | ||
+ | <TD>'''Link'''</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>Which hormone is excreted by the juxtaglomerular cells in response to a fall in blood pressure?</TD> | ||
+ | <TD> | ||
+ | <FONT COLOR="#FFFFFF">Renin</FONT></TD> | ||
+ | <TD>[[Renin Angiotensin Aldosterone System|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>What does ACE do?</TD> | ||
+ | <TD> | ||
+ | <FONT COLOR="#FFFFFF">Converts angiotensin 1 to angiotensin 2</FONT></TD> | ||
+ | <TD>[[Renin Angiotensin Aldosterone System#|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>Which receptors does angiotensin 2 act on to cause the release of aldosterone?</TD> | ||
+ | <TD> | ||
+ | <FONT COLOR="#FFFFFF"> | ||
+ | AT1</FONT></TD> | ||
+ | <TD>[[Renin Angiotensin Aldosterone System|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>How does angiotensin 2 effect GFR?</TD> | ||
+ | <TD> | ||
+ | <FONT COLOR="#FFFFFF"> | ||
+ | Contracts the efferent arteriole to increase GFR when blood pressure drops</FONT></TD> | ||
+ | <TD>[[Renin Angiotensin Aldosterone System#Effects of Angiotensin 2 on GFR|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>What three things stimulate the release of aldosterone?</TD> | ||
+ | <TD> | ||
+ | <FONT COLOR="#FFFFFF"> | ||
+ | * Corticotropin (ACTH) | ||
+ | * Angiotensin 2 | ||
+ | * K<sup>+</sup></FONT></TD> | ||
+ | <TD>[[Aldosterone#Release|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>What are its effects on sodium?</TD> | ||
+ | <TD> | ||
+ | <FONT COLOR="#FFFFFF">Increases the reabsorption of sodium from the distal tubule and collecting duct</FONT></TD> | ||
+ | <TD>[[Aldosterone#Action|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>How does it achieve these effects?</TD> | ||
+ | <TD> | ||
+ | <FONT COLOR="#FFFFFF">Increases the synthesis of sodium transporters and increases the supply of ATP to them by altering gene transcription</FONT></TD> | ||
+ | <TD>[[Aldosterone#Action|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>Where is atrial natriutretic peptide secreted from?</TD> | ||
+ | <TD> | ||
+ | <FONT COLOR="#FFFFFF">Atrial walls</FONT></TD> | ||
+ | <TD>[[Atrial Natriuretic Peptide|Link]]</TD> | ||
+ | </TR> | ||
+ | <TR> | ||
+ | <TD>Whats it's effect?</TD> | ||
+ | <TD> | ||
+ | <FONT COLOR="#FFFFFF">Increases the excretion of water by reducing the reuptake of sodium.</FONT></TD> | ||
+ | <TD>[[Atrial Natriuretic Peptide|Link]]</TD> | ||
+ | </TR> | ||
+ | </table> | ||
+ | |||
+ | |||
+ | [[Category:Kidney Hormonal Regulators]][[Category:Endocrine System - Anatomy & Physiology]][[Category:Urinary System Anatomy & Physiology Flashcards]] |
Latest revision as of 17:21, 21 June 2011
Use the mouse to highlight the answers. They are written in white
Question | Answer | Link |
Which hormone is excreted by the juxtaglomerular cells in response to a fall in blood pressure? | Renin | Link |
What does ACE do? | Converts angiotensin 1 to angiotensin 2 | Link |
Which receptors does angiotensin 2 act on to cause the release of aldosterone? |
AT1 |
Link |
How does angiotensin 2 effect GFR? |
Contracts the efferent arteriole to increase GFR when blood pressure drops |
Link |
What three things stimulate the release of aldosterone? |
|
Link |
What are its effects on sodium? | Increases the reabsorption of sodium from the distal tubule and collecting duct | Link |
How does it achieve these effects? | Increases the synthesis of sodium transporters and increases the supply of ATP to them by altering gene transcription | Link |
Where is atrial natriutretic peptide secreted from? | Atrial walls | Link |
Whats it's effect? | Increases the excretion of water by reducing the reuptake of sodium. | Link |