Difference between revisions of "Frohlich's Adiposogenital Syndrome"

Revision as of 17:11, 5 September 2008

Clinical signs due to a hypothalamic dysfunction.

Decresaed synthesis and release of GnRH means that the pituitary is not stimulated to release FSH or LH at normal levels. Clincally seen as:

Hypothalamus damage also allows incresaed appetite and the animal will be:

Compression of local structures also leads to:

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+{{toplink
+|backcolour = FAFAD2
+|linkpage =Endocrine System - Pathology
+|linktext =Endocrine System
+|maplink = Endocrine System (Content Map) - Pathology
+|pagetype =Pathology
+|sublink1=Pituitary Gland - Pathology
+|subtext1=PITUITARY GLAND
+}}
+<br>
 Clinical signs due to a hypothalamic dysfunction.
-Decreased synthesis and release of GnRH means that the pituitary is not stimulated to release FSH or LH at normal levels.  Clinically seen as:
+Decresaed synthesis and release of GnRH means that the pituitary is not stimulated to release FSH or LH at normal levels.  Clincally seen as:
 *Small, atrophic genitals.
 *Feminisation in the male due to lower testosterone release.
-Hypothalamus damage also allows increased appetite and the animal may be obese, with excess deposition of subcutaneous fat.
+Hypothalamus damage also allows incresaed appetite and the animal will be:
+*Obese.
+*Excess deposition of sub-cut fat.
 Compression of local structures also leads to:
-*Mental retardation.
+*Mental retartdation.
 *Diabetes insipidus.
 *Visual impairment.
-[[Category:To Do - Reproductive]]
-[[Category:Hypothalamus - Pathology]]
-[[Category:Reproductive System - Pathology]]