Difference between revisions of "Category:Enteropathogenic and Enterotoxaemic Clostridia"
Jump to navigation
Jump to search
Line 15: | Line 15: | ||
− | ===''[[Clostridium perfringens type | + | ===''[[Clostridium perfringens type D]]=== |
Line 26: | Line 26: | ||
===''C. perfringens'' type D=== | ===''C. perfringens'' type D=== | ||
− | + | ||
− | |||
− | |||
− | |||
− | |||
− | |||
− | |||
− | |||
− | |||
− | |||
− | |||
Revision as of 11:08, 12 May 2010
- General:
- Clostridium perfringens types B, C and D
- Found in soil, feaces and intestinal tract
- Survive in soil as spores
- Husbandry, changes in diet and environment predispose to proliferation in the intestine
- Abrupt changes to rich diets and intestinal hypomotility due to overeating
- Pathogenesis and pathogenicity:
- Clostridial replication and overgrowth in the interstinal tract of sheep
- Production of potent exotoxins which cause local and systemic effects of enterotoxaemia
- Type of toxins produced determine clinical syndrome
- Haemolysins, collagenases and hyaluronidases also produced
Clostridium perfringens type D
C. perfringens type C
C. perfringens type D
C. perfringens type E
- Enteritis in rabbits, haemorrhagic enteritis in calves
- ALpha and iota toxins
Treatment and control of enterotoxaemic infections
- Hyperimmune serum
- Vaccination - vaccinate ewes with toxoid 6 weeks before lambing to allow passive protection of lambs
- Vaccination of lambs with toxoid before 2 months of age to protect against pulpy kidney
- Avoid sudden dietary changes
Pages in category "Enteropathogenic and Enterotoxaemic Clostridia"
The following 5 pages are in this category, out of 5 total.